Links to Articles, Sites, and Studies Exploring the Medicinal Use of Marijuana

This is just a small collection of the material I’ve been reading on Cannabis and it’s potential as a cure for numerous diseases. There is much more out there.

Remember, the best research are the ones you do on your own. Take 5 seconds, google “Cannabis Cures Cancer,” and see for yourself what you find.

National Cancer Institute Website

http://www.cancer.gov/cancertopics/pdq/cam/cannabis/healthprofessional/page4

http://www.cancer.org/treatment/

Yes, you are reading that correctly. That is the official government website on cancer. If government websites such as the National Cancer Institute state numerous medicinal benefits of cannabis, then why is it a Schedule I drug? Here’s a link to the justice.gov website and the definition of drug scheduling.

Schedule I

Schedule I drugs, substances, or chemicals are defined as drugs with no currently accepted medical use and a high potential for abuse. Schedule I drugs are the most dangerous drugs of all the drug schedules with potentially severe psychological or physical dependence. Some examples of Schedule I drugs are:

heroin, lysergic acid diethylamide (LSD), marijuana (cannabis), 3,4-methylenedioxymethamphetamine (ecstasy), methaqualone, and peyote”

Various Publications

http://www.phoenixtearsfoundation.com/scientific-studies/ (Collection of articles and studies).

https://cannajournal.com/2013/12/19/an-overview-of-the-endocannabinoid-system-and-cannabinoids-how-cannabis-cures-cancer/ – An overview of the endocannabinoid system and cannabinoids – the science behind how marijuana cures cancer and more.

https://cannajournal.com/2013/12/26/consuming-raw-cannabis-to-cure-cancer-cannabis-as-a-plant-and-vegetable-you-dont-get-high/ – Collection of recordings, documentaries, and links discussing consuming cannabis in it’s raw form so that you don’t receive the psychoactive effects.

http://buelahman.wordpress.com/2013/07/30/34-medical-studies-proving-cannabis-cures-cancer/ (Collection of 34 studies that prove cannabis cures various types of cancer).

http://www.sciencedaily.com/releases/2007/04/070417193338.htm – Marijuana Cuts Lung Cancer Tumor Growth in Half.

Research Studies Archived at the US National Library of Medicine

This is not an exhaustive list. There are literally hundreds more. Please note the dates in which the studies were conducted. One must ask, “why isn’t there more publicity surrounding this?”

And isn’t it weird that both the Federal Government and Pharmaceutical companies have been submitting patents on synthetic versions of cannabis extracts for use on various ailments over the past decade? Despite this, Cannabis Prohibition goes on…

http://www.ncbi.nlm.nih.gov/pubmed/14570037 – 2003 study. Cannabinoids as potential anticancer agents.

http://www.ncbi.nlm.nih.gov/pubmed/12648025 – 2003 study. Pharmacokinetics and pharmacodynamics of cannabinoids.

http://www.ncbi.nlm.nih.gov/pubmed/19914218 – 2010 study. Cannabidiol inhibits cancer cell invasion via upregulation of tissue inhibitor of matrix metalloproteinases -1.

http://www.ncbi.nlm.nih.gov/pubmed/15026328 – 2004 study. Cannabinoids induce cancer cell proliferation via tumor necrosis factor alpha-converting enzyme (TACE/ADAM17)-mediated transactivation of the epidermal growth factor receptor.

http://www.ncbi.nlm.nih.gov/pubmed/15361550 – 2004 study. Up-regulation of cyclooxygenase-2 expression is involved in R(+)-methanandamide-induced apoptotic death of human neuroglioma cells.

http://www.ncbi.nlm.nih.gov/pubmed/19889794 – 2010 study. Antitumorigenic effects of cannabinoids beyond apoptosis.

http://www.ncbi.nlm.nih.gov/pubmed/19015962 – 2009 study. R(+)-methanandamide-induced apoptosis of human cervical carcinoma cells involves a cyclooxygenase-2-dependent pathway.

http://www.ncbi.nlm.nih.gov/pubmed/19608284 – 2009 study. The endocannabinoid system of the skin in health and disease: novel perspectives and therapeutic opportunities.

http://www.ncbi.nlm.nih.gov/pubmed/17237277 – 2007 study. HU-331, a novel cannabinoid-based anticancer topoisomerase II inhibitor.

http://www.ncbi.nlm.nih.gov/pubmed/11586361 – 2001 study. An endogenous cannabinoid (2-AG) is neuroprotective after brain injury.

http://www.ncbi.nlm.nih.gov/pubmed/14692532 – 2003 study. Gamma-irradiation enhances apoptosis induced by cannabidiol, a non-psychotropic cannabinoid, in cultured HL-60 myeloblastic leukemia cells.

http://www.ncbi.nlm.nih.gov/pubmed/16571653 – 2006 study. A cannabinoid quinone inhibits angiogenesis by targeting vascular endothelial cells.

http://www.ncbi.nlm.nih.gov/pubmed/18286801 – 2007 study. Cannabinoids in health and disease.

http://www.ncbi.nlm.nih.gov/pubmed/16250836 – 2005 study. Cannabinoids and Cancer.

http://www.ncbi.nlm.nih.gov/pubmed/17934890 – 2008 study. Delta 9-tetrahydrocannabinol inhibits cell cycle progression by downregulation of E2F1 in human glioblastoma multiforme cells.

http://www.ncbi.nlm.nih.gov/pubmed/12052046 – 2002 study. Endocannabinoids in the immune system and cancer.

http://www.ncbi.nlm.nih.gov/pubmed/19189054 – 2010 study. A metabolically stable analogue of anandamide, Met-F-AEA, inhibits human thyroid carcinoma cell lines by activation of apoptosis.

http://www.ncbi.nlm.nih.gov/pubmed/19047095 – 2008 study. Cannabinoid receptor activation induces apoptosis through tumor necrosis factor alpha-mediated ceramide de novo synthesis in colon cancer cells.

http://www.ncbi.nlm.nih.gov/pubmed/10913156 – 2000 study. Anandamide induces apoptosis in human cells via vanilloid receptors. Evidence for a protective role of cannabinoid receptors.

http://www.ncbi.nlm.nih.gov/pubmed/9653194 – 1998 study. The endogenous cannabinoid anandamide inhibits human breast cancer cell proliferation.

http://www.ncbi.nlm.nih.gov/pubmed/18088200 – 2008 study. Cannabinoids as potential new therapy for the treatment of gliomas.

http://www.ncbi.nlm.nih.gov/pubmed/16909207 – 2006 study. The non-psychoactive cannabidiol triggers caspase activation and oxidative stress in human glioma cells.

http://www.ncbi.nlm.nih.gov/pubmed/17342320 – 2007 study. Endocannabinoids as emerging suppressors of angiogenesis and tumor invasion (review).

http://www.ncbi.nlm.nih.gov/pubmed/19059457 – 2009 study. Apoptosis induced in HepG2 cells by the synthetic cannabinoid WIN: involvement of the transcription factor PPARgamma.

http://www.ncbi.nlm.nih.gov/pubmed/12723496 – 2003 study. [The endocannabinoid system as a target for the development of new drugs for cancer therapy].

http://www.ncbi.nlm.nih.gov/pubmed/19442536 – 2009 study. Cannabinoids in intestinal inflammation and cancer.

http://www.ncbi.nlm.nih.gov/pubmed/16728591 – 2006 study. Antitumor activity of plant cannabinoids with emphasis on the effect of cannabidiol on human breast carcinoma.

http://www.ncbi.nlm.nih.gov/pubmed/19539619 – 2009 study. The CB1/CB2 receptor agonist WIN-55,212-2 reduces viability of human Kaposi’s sarcoma cells in vitro.

http://www.ncbi.nlm.nih.gov/pubmed/16500647 – 2006 study. Cannabinoid derivatives induce cell death in pancreatic MIA PaCa-2 cells via a receptor-independent mechanism.

http://www.ncbi.nlm.nih.gov/pubmed/19189659 – 2008 study. The endocannabinoid anandamide neither impairs in vitro T-cell function nor induces regulatory T-cell generation.

http://www.ncbi.nlm.nih.gov/pubmed/14617682 – 2004 study. Antitumor effects of cannabidiol, a nonpsychoactive cannabinoid, on human glioma cell lines.

http://www.ncbi.nlm.nih.gov/pubmed/18938775 – 2008 study. Estrogenic induction of cannabinoid CB1 receptor in human colon cancer cell lines.

http://www.ncbi.nlm.nih.gov/pubmed/11106791 – 2000 study. Endocannabinoids and fatty acid amides in cancer, inflammation and related disorders.

http://www.ncbi.nlm.nih.gov/pubmed/19394652 – 2009 study. Pharmacological synergism between cannabinoids and paclitaxel in gastric cancer cell lines.

http://www.ncbi.nlm.nih.gov/pubmed/20336665 – 2010 study. Effect of a synthetic cannabinoid agonist on the proliferation and invasion of gastric cancer cells.

http://www.ncbi.nlm.nih.gov/pubmed/19442435 – 2009 study. Cannabinoids in the treatment of cancer.

http://www.ncbi.nlm.nih.gov/pubmed/18197164 – 2008 study. Cannabinoid 2 receptor induction by IL-12 and its potential as a therapeutic target for the treatment of anaplastic thyroid carcinoma.

http://www.ncbi.nlm.nih.gov/pubmed/16835997 – 2006 study. [Different views on the association between cannabinoids and cancer].

http://www.ncbi.nlm.nih.gov/pubmed/11903061 – 2002 study. De novo-synthesized ceramide is involved in cannabinoid-induced apoptosis.

http://www.ncbi.nlm.nih.gov/pubmed/17675107 – 2008 study. Down-regulation of tissue inhibitor of metalloproteinases-1 in gliomas: a new marker of cannabinoid antitumoral activity?

http://www.ncbi.nlm.nih.gov/pubmed/19425170 – 2009 study. Cannabinoid action induces autophagy-mediated cell death through stimulation of ER stress in human glioma cells.

http://www.ncbi.nlm.nih.gov/pubmed/18454173 – 2008 study. JunD is involved in the antiproliferative effect of Delta9-tetrahydrocannabinol on human breast cancer cells.

http://www.ncbi.nlm.nih.gov/pubmed/17065222 – 2006 study. Cannabinoid receptors as novel targets for the treatment of melanoma.

http://www.ncbi.nlm.nih.gov/pubmed/10700234 – 2000 study. Anti-tumoral action of cannabinoids: involvement of sustained ceramide accumulation and extracellular signal-regulated kinase activation.

http://www.ncbi.nlm.nih.gov/pubmed/16787257 – 2006 study. Endocannabinoids: a new family of lipid mediators involved in the regulation of neural cell development.

http://www.ncbi.nlm.nih.gov/pubmed/16139274 – 2005 study. p38 MAPK is involved in CB2 receptor-induced apoptosis of human leukaemia cells.

http://www.ncbi.nlm.nih.gov/pubmed/16624285 – 2006 study. The CB2 cannabinoid receptor signals apoptosis via ceramide-dependent activation of the mitochondrial intrinsic pathway.

http://www.ncbi.nlm.nih.gov/pubmed/16616335 – 2006 study. The stress-regulated protein p8 mediates cannabinoid-induced apoptosis of tumor cells.

http://www.ncbi.nlm.nih.gov/pubmed/11269508 – 2001 study. Control of the cell survival/death decision by cannabinoids.

http://www.ncbi.nlm.nih.gov/pubmed/19690545 – 2009 study. Inhibition of human tumour prostate PC-3 cell growth by cannabinoids R(+)-Methanandamide and JWH-015: involvement of CB2.

http://www.ncbi.nlm.nih.gov/pubmed/12511587 – 2003 study. Inhibition of skin tumor growth and angiogenesis in vivo by activation of cannabinoid receptors.

http://www.ncbi.nlm.nih.gov/pubmed/20307616 – 2010 study. Opposite changes in cannabinoid CB1 and CB2 receptor expression in human gliomas.

http://www.ncbi.nlm.nih.gov/pubmed/16818634 – 2006 study. Delta9-tetrahydrocannabinol inhibits cell cycle progression in human breast cancer cells through Cdc2 regulation.

http://www.ncbi.nlm.nih.gov/pubmed/16818650 – 2006 study. Cannabinoids induce apoptosis of pancreatic tumor cells via endoplasmic reticulum stress-related genes.

http://www.ncbi.nlm.nih.gov/pubmed/16596790– 2005 study. Effects on cell viability.

http://www.ncbi.nlm.nih.gov/pubmed/15638794 – 2005 study. Involvement of cannabinoids in cellular proliferation.

I’ll be updating this resource page as much as I can. I hope you find all this useful, educational, and even entertaining; hopefully a combination of all 3.

Related articles

One Response to Links to Articles, Sites, and Studies Exploring the Medicinal Use of Marijuana

  1. Artemis2318 says:

    I don’t understand. The government says that the studies proving the effectiveness of marijuana are still limited. Honestly, exactly how many research do they need for them to believe that marijuana is harmless and amazingly a miraculous plant that can potentially treat vast numbers of illnesses?

Leave a Reply

Fill in your details below or click an icon to log in:

WordPress.com Logo

You are commenting using your WordPress.com account. Log Out / Change )

Twitter picture

You are commenting using your Twitter account. Log Out / Change )

Facebook photo

You are commenting using your Facebook account. Log Out / Change )

Google+ photo

You are commenting using your Google+ account. Log Out / Change )

Connecting to %s